H2N2V1, Main, Exploration, bibRecord, 001E47

Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses

Identifieur interne : 001E47 ( Main/Exploration ); précédent : 001E46; suivant : 001E48

Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses

Auteurs : M. Tamura [États-Unis] ; R. G. Webster ; F. A. Ennis [États-Unis]

Source :

Journal of virology [ 0022-538X ] ; 1994.

RBID : Pascal:94-0535195

Descripteurs français

KwdFr :
- Animaux, Anticorps antiviraux (biosynthèse), Anticorps monoclonaux, Antigènes viraux, Cellules présentatrices d'antigène (immunologie), Cellules présentatrices d'antigène (microbiologie), Grippe humaine (enzymologie), Grippe humaine (immunologie), Grippe humaine (microbiologie), Humains, Lignée cellulaire, Mâle, Réactions croisées, Récepteur Fc (métabolisme), Sialidase (immunologie), Souris, Souris de lignée BALB C, Tests de neutralisation, Virus de la grippe A (), Virus de la grippe A (immunologie).
MESH :
- biosynthèse : Anticorps antiviraux.
- enzymologie : Grippe humaine.
- immunologie : Cellules présentatrices d'antigène, Grippe humaine, Sialidase, Virus de la grippe A.
- microbiologie : Cellules présentatrices d'antigène, Grippe humaine.
- métabolisme : Récepteur Fc.
Pascal (Inist)
- Animaux, Anticorps monoclonaux, Antigènes viraux, Humains, Influenzavirus A, Lignée cellulaire, Mâle, Réactions croisées, Souris, Relation hôte virus, Présentation antigène, Souris de lignée BALB C, Spécificité type, Réaction croisée, Cellule lymphoïde, Lignée P388D1, Tests de neutralisation, Virus de la grippe A.

English descriptors

KwdEn :
- Animals, Antibodies, Monoclonal, Antibodies, Viral (biosynthesis), Antigen presentation, Antigen-Presenting Cells (immunology), Antigen-Presenting Cells (microbiology), Antigens, Viral, Cell Line, Cross Reactions, Cross reaction, Host virus relation, Humans, Influenza A virus (classification), Influenza A virus (immunology), Influenza, Human (enzymology), Influenza, Human (immunology), Influenza, Human (microbiology), Influenzavirus A, Lymphoid cell, Male, Mice, Mice, Inbred BALB C, Mouse, Neuraminidase (immunology), Neutralization Tests, Receptors, Fc (metabolism), Type specificity.
MESH :
- chemical , biosynthesis : Antibodies, Viral.
- chemical , immunology : Neuraminidase.
- chemical , metabolism : Receptors, Fc.
- chemical : Antibodies, Monoclonal, Antigens, Viral.
- classification : Influenza A virus.
- enzymology : Influenza, Human.
- immunology : Antigen-Presenting Cells, Influenza A virus, Influenza, Human.
- microbiology : Antigen-Presenting Cells, Influenza, Human.
- Animals, Cell Line, Cross Reactions, Humans, Male, Mice, Mice, Inbred BALB C, Neutralization Tests.

Abstract

Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses

Affiliations:

États-Unis

Le document en format XML

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<term>Antibodies, Monoclonal</term>
<term>Antibodies, Viral (biosynthesis)</term>
<term>Antigen presentation</term>
<term>Antigen-Presenting Cells (immunology)</term>
<term>Antigen-Presenting Cells (microbiology)</term>
<term>Antigens, Viral</term>
<term>Cell Line</term>
<term>Cross Reactions</term>
<term>Cross reaction</term>
<term>Host virus relation</term>
<term>Humans</term>
<term>Influenza A virus (classification)</term>
<term>Influenza A virus (immunology)</term>
<term>Influenza, Human (enzymology)</term>
<term>Influenza, Human (immunology)</term>
<term>Influenza, Human (microbiology)</term>
<term>Influenzavirus A</term>
<term>Lymphoid cell</term>
<term>Male</term>
<term>Mice</term>
<term>Mice, Inbred BALB C</term>
<term>Mouse</term>
<term>Neuraminidase (immunology)</term>
<term>Neutralization Tests</term>
<term>Receptors, Fc (metabolism)</term>
<term>Type specificity</term>
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<term>Anticorps antiviraux (biosynthèse)</term>
<term>Anticorps monoclonaux</term>
<term>Antigènes viraux</term>
<term>Cellules présentatrices d'antigène (immunologie)</term>
<term>Cellules présentatrices d'antigène (microbiologie)</term>
<term>Grippe humaine (enzymologie)</term>
<term>Grippe humaine (immunologie)</term>
<term>Grippe humaine (microbiologie)</term>
<term>Humains</term>
<term>Lignée cellulaire</term>
<term>Mâle</term>
<term>Réactions croisées</term>
<term>Récepteur Fc (métabolisme)</term>
<term>Sialidase (immunologie)</term>
<term>Souris</term>
<term>Souris de lignée BALB C</term>
<term>Tests de neutralisation</term>
<term>Virus de la grippe A ()</term>
<term>Virus de la grippe A (immunologie)</term>
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</keywords>
<keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Receptors, Fc</term>
</keywords>
<keywords scheme="MESH" type="chemical" xml:lang="en"><term>Antibodies, Monoclonal</term>
<term>Antigens, Viral</term>
</keywords>
<keywords scheme="MESH" qualifier="biosynthèse" xml:lang="fr"><term>Anticorps antiviraux</term>
</keywords>
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</keywords>
<keywords scheme="MESH" qualifier="enzymologie" xml:lang="fr"><term>Grippe humaine</term>
</keywords>
<keywords scheme="MESH" qualifier="enzymology" xml:lang="en"><term>Influenza, Human</term>
</keywords>
<keywords scheme="MESH" qualifier="immunologie" xml:lang="fr"><term>Cellules présentatrices d'antigène</term>
<term>Grippe humaine</term>
<term>Sialidase</term>
<term>Virus de la grippe A</term>
</keywords>
<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Antigen-Presenting Cells</term>
<term>Influenza A virus</term>
<term>Influenza, Human</term>
</keywords>
<keywords scheme="MESH" qualifier="microbiologie" xml:lang="fr"><term>Cellules présentatrices d'antigène</term>
<term>Grippe humaine</term>
</keywords>
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<term>Influenza, Human</term>
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</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Cell Line</term>
<term>Cross Reactions</term>
<term>Humans</term>
<term>Male</term>
<term>Mice</term>
<term>Mice, Inbred BALB C</term>
<term>Neutralization Tests</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Animaux</term>
<term>Anticorps monoclonaux</term>
<term>Antigènes viraux</term>
<term>Humains</term>
<term>Influenzavirus A</term>
<term>Lignée cellulaire</term>
<term>Mâle</term>
<term>Réactions croisées</term>
<term>Souris</term>
<term>Relation hôte virus</term>
<term>Présentation antigène</term>
<term>Souris de lignée BALB C</term>
<term>Spécificité type</term>
<term>Réaction croisée</term>
<term>Cellule lymphoïde</term>
<term>Lignée P388D1</term>
<term>Tests de neutralisation</term>
<term>Virus de la grippe A</term>
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<front><div type="abstract" xml:lang="en">Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses</div>
</front>
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<tree><noCountry><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R. G." last="Webster">R. G. Webster</name>
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Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses

Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

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